A composite variable is a combination of multiple variables. It is used to measure multidimensional aspects that are difficult to observe. | - Entertainment
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Frequently Asked QuestionsWhat are the 10 types of variables in research. The 10 types of variables in research are: - Independent
- Confounding
- Categorical
- Extraneous.
What is an independent variable?An independent variable, often termed the predictor or explanatory variable, is the variable manipulated or categorized in an experiment to observe its effect on another variable, called the dependent variable. It’s the presumed cause in a cause-and-effect relationship, determining if changes in it produce changes in the observed outcome. What is a variable?In research, a variable is any attribute, quantity, or characteristic that can be measured or counted. It can take on various values, making it “variable.” Variables can be classified as independent (manipulated), dependent (observed outcome), or control (kept constant). They form the foundation for hypotheses, observations, and data analysis in studies. What is a dependent variable?A dependent variable is the outcome or response being studied in an experiment or investigation. It’s what researchers measure to determine the effect of changes in the independent variable. In a cause-and-effect relationship, the dependent variable is presumed to be influenced or caused by the independent variable. What is a variable in programming?In programming, a variable is a symbolic name for a storage location that holds data or values. It allows data storage and retrieval for computational operations. Variables have types, like integer or string, determining the nature of data they can hold. They’re fundamental in manipulating and processing information in software. What is a control variable?A control variable in research is a factor that’s kept constant to ensure that it doesn’t influence the outcome. By controlling these variables, researchers can isolate the effects of the independent variable on the dependent variable, ensuring that other factors don’t skew the results or introduce bias into the experiment. What is a controlled variable in science?In science, a controlled variable is a factor that remains constant throughout an experiment. It ensures that any observed changes in the dependent variable are solely due to the independent variable, not other factors. By keeping controlled variables consistent, researchers can maintain experiment validity and accurately assess cause-and-effect relationships. How many independent variables should an investigation have?Ideally, an investigation should have one independent variable to clearly establish cause-and-effect relationships. Manipulating multiple independent variables simultaneously can complicate data interpretation. However, in advanced research, experiments with multiple independent variables (factorial designs) are used, but they require careful planning to understand interactions between variables. You May Also LikeDisadvantages of primary research – It can be expensive, time-consuming and take a long time to complete if it involves face-to-face contact with customers. A confounding variable can potentially affect both the suspected cause and the suspected effect. Here is all you need to know about accounting for confounding variables in research. This article presents the key advantages and disadvantages of secondary research so you can select the most appropriate research approach for your study. USEFUL LINKS LEARNING RESOURCES ![what is an outcome variable in research researchprospect-reviews-trust-site](https://www.researchprospect.com/wp-content/uploads/2023/06/researchprospect-reviews-trust-site.webp) COMPANY DETAILS ![what is an outcome variable in research Research-Prospect-Writing-Service](https://www.researchprospect.com/wp-content/uploads/2021/08/Research-Prospect-Writing-Service.webp) Doing Quantitative Research with Outcome Measures- First Online: 24 December 2020
Cite this chapter![what is an outcome variable in research what is an outcome variable in research](https://media.springernature.com/w72/springer-static/cover-hires/book/978-3-030-55127-8?as=webp) - Charlie Duncan 2 &
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2416 Accesses 1 Citations This chapter explores some of the contextual factors and the key drivers shaping the movement towards routine measurements. It touches upon some underlying philosophies behind some key drivers and how they differentially shape the way in which outcome measurement may be implemented. It also considers the body of research which demonstrates that while different therapeutic approaches are broadly similar in their outcomes, at a service and practitioner level there is considerable variance. We provide examples of outcome measurement from research and practice settings, inviting you to consider options of engaging with measurement of outcome in therapy settings to both learn and evidence effective prac tice This is a preview of subscription content, log in via an institution to check access. Access this chapter- Available as PDF
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Tax calculation will be finalised at checkout Purchases are for personal use only Institutional subscriptions BACP. (2018a). Ethical Framework for the Counselling Professions . Available at: https://www.bacp.co.uk/events-and-resources/ethics-and-standards/ethical-framework-for-the-counselling-professions/. BACP. (2018b). Ethical Guidelines for Research in the Counselling Professions . Available at: https://www.bacp.co.uk/events-and-resources/research/publications/ethical-guidelines-for-research-in-the-counselling-professions /. Börjesson, S., & Boström, P. K. (2019). “I want to know what it is used for”: Clients’ perspectives on completing a routine outcome measure (ROM) while undergoing psychotherapy. Psychotherapy Research . https://doi.org/10.1080/10503307.2019.1630780 . Boynton, P. M., & Greenhalgh, T. (2004). Selecting, designing, and developing your questionnaire. BMJ, 328 (7451), 1312–1315. https://doi.org/10.1136/bmj.328.7451.1312 . Article Google Scholar Brown, J., Dreis, S., & Nace, D. K. (1999). What really makes a difference in psychotherapy outcome? Why does managed care want to know? In M. A. Hubble, B. L. Duncan, & S. D. Miller (Eds.), The heart and soul of change: What works in therapy (pp. 389–406). Washington, DC, US: American Psychological Association. Chapter Google Scholar Chow, D., Miller, S. D., Seidel, J. A., Kane, R. T., Thornton, J., & Andrews, W. P. (2015). The role of deliberate practice in the development of highly effective psychotherapists. Psychotherapy, 52 (3), 337–345. http://dx.doi.org/10.1037/pst0000015 Firth, N., Saxon, D., Stiles, W. B., & Barkham, M. (2019) Therapist and clinic effects in psychotherapy: a three-level model of outcome variability. Journal of Consulting and Clinical Psychology , 87(4), 345–356. Google Scholar Goldberg, S. B., Rousmaniere, T., Miller, S. D., Whipple, J., Nielsen, S. L., et al. (2016). Do psychotherapists improve with time and experience? A longitudinal analysis of outcomes in a clinical setting. Journal of Counseling Psychology, 63 (1), 1–11. Hannan, C., Lambert, M. J., Harmon, C., Nielsen, S. L., Smart, D. W., et al. (2005). A lab test and algorithms for identifying clients at risk for treatment failure. Journal of Clinical Psychology, 61 (2), 155–163. Heale, R., & Twycross, A. (2015). Validity and reliability in quantitative studies. Evidence-Based Nursing, 18 , 66–67. Howard, K. I., Kopta, S. M., Krause, M. S., & Orlinsky, D. E. (1986). The dose-effect relationship in psychotherapy. American Psychologist, 41 (2), 159–164. Jefford, M., Stockler, M.R., Tattersa, M. (2003) Outcomes research: what is it and why does it matter? Internal Medicine Journal . 33(3):110–8. doi: 10.1046/j.1445-5994.2003.00302.x. Lambert, M. J., Harmon, C., Slade, K., Whipple, J. L., & Hawkins, E. J. (2005). Providing feedback to psychotherapists on their patients’ progress: Clinical results and practice suggestions. Journal of Clinical Psychology, 61 (2), 165–174. Lambert, M. J. (2013). Outcome in psychotherapy: The past and important advances. Psychotherapy, 50 (1), 42–51. https://doi.org/10.1037/a0030682 Law, D. (2018). Goals and goal-based outcomes (GBOs): Goal progress chart. Available at: https://goalsintherapycom.files.wordpress.com/2018/03/gbo-version-2-march-2018-final.pdf Marsella, A. J. (2003). Cultural Aspects of Depressive Experience and Disorders. Online Readings in Psychology and Culture, 10 (2). https://doi.org/10.9707/2307-0919.1081 . Miller, S. D., Duncan, B. L., Sorrell, R., Brown, G. S., & Chalk, M. B. (2006). Using outcome to inform therapy practice. Journal of Brief Therapy , 5(1), 5–22. Okiishi, J., Lambert, M., Nielsen, S. L., & Ogles, B. M. (2003). Waiting for supershrink: An empirical analysis of therapist effects. Clinical Psychology and Psychotherapy, 10 (6), 361–373. Stafford, M. R., Cooper, M., Barkham, M., Beecham, J., Bower, P., Cromarty, K., et al. (2018). Effectiveness and cost-effectiveness of humanistic counselling in schools for young people with emotional distress (ETHOS): study protocol for a randomised controlled trial. Trials, 19 , 175. https://doi.org/10.1186/s13063-018-2538-2 . Van Rijn, B. (2020). Evaluating Our Practice. In S. Bager-Charleson (Ed.), Reflective Practice and Personal Development in the field of Therapy . London: Sage. Wampold, B. E., & Imel, Z. E. (2015). The great psychotherapy debate: The evidence for what makes psychotherapy work (Second Edition) . New York, New York: Routledge. https://doi.org/10.4324/9780203582015 . Book Google Scholar Whipple, J. L., Lambert, M. J., Vermeersch, D. A., Smart, D. W., Nielsen, S. L., & Hawkins, E. J. (2003). Improving the effects of psychotherapy: The use of early identification of treatment and problem-solving strategies in routine practice. Journal of Counseling Psychology, 50 (1), 59–68. Download references Author informationAuthors and affiliations. (BACP) The British Association for Counselling and Psychotherapy, Lutterworth, Leicestershire, UK Charlie Duncan & Barry McInnes You can also search for this author in PubMed Google Scholar Corresponding authorCorrespondence to Charlie Duncan . Editor informationEditors and affiliations. Metanoia Institute, London, UK Sofie Bager-Charleson & Alistair McBeath & Rights and permissionsReprints and permissions Copyright information© 2020 The Author(s) About this chapterDuncan, C., McInnes, B. (2020). Doing Quantitative Research with Outcome Measures. In: Bager-Charleson, S., McBeath, A. (eds) Enjoying Research in Counselling and Psychotherapy. Palgrave Macmillan, Cham. https://doi.org/10.1007/978-3-030-55127-8_11 Download citationDOI : https://doi.org/10.1007/978-3-030-55127-8_11 Published : 24 December 2020 Publisher Name : Palgrave Macmillan, Cham Print ISBN : 978-3-030-55126-1 Online ISBN : 978-3-030-55127-8 eBook Packages : Behavioral Science and Psychology Behavioral Science and Psychology (R0) Share this chapterAnyone you share the following link with will be able to read this content: Sorry, a shareable link is not currently available for this article. Provided by the Springer Nature SharedIt content-sharing initiative Policies and ethics - Find a journal
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Outcome variables - Meaning & DefinitionWhat is outcome variables. Outcome variables are usually the dependent variables which are observed and measured by changing independent variables. These variables determine the effect of the cause (independent) variables when changed for different values. The dependent variables are the outcomes of the experiments determining what was caused or what changed as a result of the study. For a simple example, the marks a student obtains in an exam is a result of the hard word measured in the number of hours put behind studying and the intelligence measured in IQ are the independent variables. The marks obtained thus represents the dependent or outcome variable. When the values of the independent variables are changed, the marks may or may not change and hence dependent variables are dependent on the independent variables while the opposite is implausible i.e., when the marks are changed, it doesn’t change the number of hours of study or the IQ of the student. The response variable is also called as the dependent variable because it depends on the causal factor, the independent variable. Depending on the various input values of the experimental variables, the responses are recorded. This article has been researched & authored by the Business Concepts Team . It has been reviewed & published by the MBA Skool Team. The content on MBA Skool has been created for educational & academic purpose only. Browse the definition and meaning of more similar terms. The Management Dictionary covers over 1800 business concepts from 5 categories. Continue Reading: - Class Interval
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![what is an outcome variable in research FB Page](https://www.mbaskool.com/images/fb_ico.png) Research Hypothesis In Psychology: Types, & ExamplesSaul Mcleod, PhD Editor-in-Chief for Simply Psychology BSc (Hons) Psychology, MRes, PhD, University of Manchester Saul Mcleod, PhD., is a qualified psychology teacher with over 18 years of experience in further and higher education. He has been published in peer-reviewed journals, including the Journal of Clinical Psychology. Learn about our Editorial Process Olivia Guy-Evans, MSc Associate Editor for Simply Psychology BSc (Hons) Psychology, MSc Psychology of Education Olivia Guy-Evans is a writer and associate editor for Simply Psychology. She has previously worked in healthcare and educational sectors. On This Page: A research hypothesis, in its plural form “hypotheses,” is a specific, testable prediction about the anticipated results of a study, established at its outset. It is a key component of the scientific method . Hypotheses connect theory to data and guide the research process towards expanding scientific understanding Some key points about hypotheses:- A hypothesis expresses an expected pattern or relationship. It connects the variables under investigation.
- It is stated in clear, precise terms before any data collection or analysis occurs. This makes the hypothesis testable.
- A hypothesis must be falsifiable. It should be possible, even if unlikely in practice, to collect data that disconfirms rather than supports the hypothesis.
- Hypotheses guide research. Scientists design studies to explicitly evaluate hypotheses about how nature works.
- For a hypothesis to be valid, it must be testable against empirical evidence. The evidence can then confirm or disprove the testable predictions.
- Hypotheses are informed by background knowledge and observation, but go beyond what is already known to propose an explanation of how or why something occurs.
Predictions typically arise from a thorough knowledge of the research literature, curiosity about real-world problems or implications, and integrating this to advance theory. They build on existing literature while providing new insight. Types of Research HypothesesAlternative hypothesis. The research hypothesis is often called the alternative or experimental hypothesis in experimental research. It typically suggests a potential relationship between two key variables: the independent variable, which the researcher manipulates, and the dependent variable, which is measured based on those changes. The alternative hypothesis states a relationship exists between the two variables being studied (one variable affects the other). A hypothesis is a testable statement or prediction about the relationship between two or more variables. It is a key component of the scientific method. Some key points about hypotheses: - Important hypotheses lead to predictions that can be tested empirically. The evidence can then confirm or disprove the testable predictions.
In summary, a hypothesis is a precise, testable statement of what researchers expect to happen in a study and why. Hypotheses connect theory to data and guide the research process towards expanding scientific understanding. An experimental hypothesis predicts what change(s) will occur in the dependent variable when the independent variable is manipulated. It states that the results are not due to chance and are significant in supporting the theory being investigated. The alternative hypothesis can be directional, indicating a specific direction of the effect, or non-directional, suggesting a difference without specifying its nature. It’s what researchers aim to support or demonstrate through their study. Null HypothesisThe null hypothesis states no relationship exists between the two variables being studied (one variable does not affect the other). There will be no changes in the dependent variable due to manipulating the independent variable. It states results are due to chance and are not significant in supporting the idea being investigated. The null hypothesis, positing no effect or relationship, is a foundational contrast to the research hypothesis in scientific inquiry. It establishes a baseline for statistical testing, promoting objectivity by initiating research from a neutral stance. Many statistical methods are tailored to test the null hypothesis, determining the likelihood of observed results if no true effect exists. This dual-hypothesis approach provides clarity, ensuring that research intentions are explicit, and fosters consistency across scientific studies, enhancing the standardization and interpretability of research outcomes. Nondirectional HypothesisA non-directional hypothesis, also known as a two-tailed hypothesis, predicts that there is a difference or relationship between two variables but does not specify the direction of this relationship. It merely indicates that a change or effect will occur without predicting which group will have higher or lower values. For example, “There is a difference in performance between Group A and Group B” is a non-directional hypothesis. Directional HypothesisA directional (one-tailed) hypothesis predicts the nature of the effect of the independent variable on the dependent variable. It predicts in which direction the change will take place. (i.e., greater, smaller, less, more) It specifies whether one variable is greater, lesser, or different from another, rather than just indicating that there’s a difference without specifying its nature. For example, “Exercise increases weight loss” is a directional hypothesis. ![Research Hypothesis In Psychology: Types, & Examples 1 hypothesis](https://www.simplypsychology.org/wp-content/uploads/hypothesis.jpg) FalsifiabilityThe Falsification Principle, proposed by Karl Popper , is a way of demarcating science from non-science. It suggests that for a theory or hypothesis to be considered scientific, it must be testable and irrefutable. Falsifiability emphasizes that scientific claims shouldn’t just be confirmable but should also have the potential to be proven wrong. It means that there should exist some potential evidence or experiment that could prove the proposition false. However many confirming instances exist for a theory, it only takes one counter observation to falsify it. For example, the hypothesis that “all swans are white,” can be falsified by observing a black swan. For Popper, science should attempt to disprove a theory rather than attempt to continually provide evidence to support a research hypothesis. Can a Hypothesis be Proven?Hypotheses make probabilistic predictions. They state the expected outcome if a particular relationship exists. However, a study result supporting a hypothesis does not definitively prove it is true. All studies have limitations. There may be unknown confounding factors or issues that limit the certainty of conclusions. Additional studies may yield different results. In science, hypotheses can realistically only be supported with some degree of confidence, not proven. The process of science is to incrementally accumulate evidence for and against hypothesized relationships in an ongoing pursuit of better models and explanations that best fit the empirical data. But hypotheses remain open to revision and rejection if that is where the evidence leads. - Disproving a hypothesis is definitive. Solid disconfirmatory evidence will falsify a hypothesis and require altering or discarding it based on the evidence.
- However, confirming evidence is always open to revision. Other explanations may account for the same results, and additional or contradictory evidence may emerge over time.
We can never 100% prove the alternative hypothesis. Instead, we see if we can disprove, or reject the null hypothesis. If we reject the null hypothesis, this doesn’t mean that our alternative hypothesis is correct but does support the alternative/experimental hypothesis. Upon analysis of the results, an alternative hypothesis can be rejected or supported, but it can never be proven to be correct. We must avoid any reference to results proving a theory as this implies 100% certainty, and there is always a chance that evidence may exist which could refute a theory. How to Write a Hypothesis- Identify variables . The researcher manipulates the independent variable and the dependent variable is the measured outcome.
- Operationalized the variables being investigated . Operationalization of a hypothesis refers to the process of making the variables physically measurable or testable, e.g. if you are about to study aggression, you might count the number of punches given by participants.
- Decide on a direction for your prediction . If there is evidence in the literature to support a specific effect of the independent variable on the dependent variable, write a directional (one-tailed) hypothesis. If there are limited or ambiguous findings in the literature regarding the effect of the independent variable on the dependent variable, write a non-directional (two-tailed) hypothesis.
- Make it Testable : Ensure your hypothesis can be tested through experimentation or observation. It should be possible to prove it false (principle of falsifiability).
- Clear & concise language . A strong hypothesis is concise (typically one to two sentences long), and formulated using clear and straightforward language, ensuring it’s easily understood and testable.
Consider a hypothesis many teachers might subscribe to: students work better on Monday morning than on Friday afternoon (IV=Day, DV= Standard of work). Now, if we decide to study this by giving the same group of students a lesson on a Monday morning and a Friday afternoon and then measuring their immediate recall of the material covered in each session, we would end up with the following: - The alternative hypothesis states that students will recall significantly more information on a Monday morning than on a Friday afternoon.
- The null hypothesis states that there will be no significant difference in the amount recalled on a Monday morning compared to a Friday afternoon. Any difference will be due to chance or confounding factors.
More Examples- Memory : Participants exposed to classical music during study sessions will recall more items from a list than those who studied in silence.
- Social Psychology : Individuals who frequently engage in social media use will report higher levels of perceived social isolation compared to those who use it infrequently.
- Developmental Psychology : Children who engage in regular imaginative play have better problem-solving skills than those who don’t.
- Clinical Psychology : Cognitive-behavioral therapy will be more effective in reducing symptoms of anxiety over a 6-month period compared to traditional talk therapy.
- Cognitive Psychology : Individuals who multitask between various electronic devices will have shorter attention spans on focused tasks than those who single-task.
- Health Psychology : Patients who practice mindfulness meditation will experience lower levels of chronic pain compared to those who don’t meditate.
- Organizational Psychology : Employees in open-plan offices will report higher levels of stress than those in private offices.
- Behavioral Psychology : Rats rewarded with food after pressing a lever will press it more frequently than rats who receive no reward.
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The impact of food insecurity on health outcomes: empirical evidence from sub-Saharan African countries- Sisay Demissew Beyene ORCID: orcid.org/0000-0001-7347-4168 1
BMC Public Health volume 23 , Article number: 338 ( 2023 ) Cite this article 25k Accesses 19 Citations 7 Altmetric Metrics details Food insecurity adversely affects human health, which means food security and nutrition are crucial to improving people’s health outcomes. Both food insecurity and health outcomes are the policy and agenda of the 2030 Sustainable Development Goals (SDGs). However, there is a lack of macro-level empirical studies (Macro-level study means studies at the broadest level using variables that represent a given country or the whole population of a country or economy as a whole. For example, if the urban population (% of the total population) of XYZ country is 30%, it is used as a proxy variable to represent represent country's urbanization level. Empirical study implies studies that employ the econometrics method, which is the application of math and statistics.) concerning the relationship between food insecurity and health outcomes in sub-Saharan African (SSA) countries though the region is highly affected by food insecurity and its related health problems. Therefore, this study aims to examine the impact of food insecurity on life expectancy and infant mortality in SSA countries. The study was conducted for the whole population of 31 sampled SSA countries selected based on data availability. The study uses secondary data collected online from the databases of the United Nations Development Programme (UNDP), the Food and Agricultural Organization (FAO), and the World Bank (WB). The study uses yearly balanced data from 2001 to 2018. This study employs a multicountry panel data analysis and several estimation techniques; it employs Driscoll-Kraay standard errors (DKSE), a generalized method of momentum (GMM), fixed effects (FE), and the Granger causality test. A 1% increment in people’s prevalence for undernourishment reduces their life expectancy by 0.00348 percentage points (PPs). However, life expectancy rises by 0.00317 PPs with every 1% increase in average dietary energy supply. A 1% rise in the prevalence of undernourishment increases infant mortality by 0.0119 PPs. However, a 1% increment in average dietary energy supply reduces infant mortality by 0.0139 PPs. ConclusionsFood insecurity harms the health status of SSA countries, but food security impacts in the reverse direction. This implies that to meet SDG 3.2, SSA should ensure food security. Peer Review reports Food security is essential to people’s health and well-being [ 1 ]. Further, the World Health Organization (WHO) argues that health is wealth and poor health is an integral part of poverty; governments should actively seek to preserve their people’s lives and reduce the incidence of unnecessary mortality and avoidable illnesses [ 2 ]. However, lack of food is one of the factors which affect health outcomes. Concerning this, the Food Research and Action Center noted that the social determinants of health, such as poverty and food insecurity, are associated with some of the most severe and costly health problems in a nation [ 3 ]. According to the FAO, the International Fund for Agricultural Development (IFAD), and the World Food Programme (WFP), food insecurity is defined as "A situation that exists when people lack secure access to sufficient amounts of safe and nutritious food for normal growth and development and an active and healthy life" ([ 4 ]; p50). It is generally believed that food security and nutrition are crucial to improving human health and development. Studies show that millions of people live in food insecurity, which is one of the main risks to human health. Around one in four people globally (1.9 billion people) were moderately or severely food insecure in 2017 and the greatest numbers were in SSA and South Asia. Around 9.2% of the world's population was severely food insecure in 2018. Food insecurity is highest in SSA countries, where nearly one-third are defined as severely insecure [ 5 ]. Similarly, 11% (820 million) of the world's population was undernourished in 2018, and SSA countries still share a substantial amount [ 5 ]. Even though globally the number of people affected by hunger has been decreasing since 1990, in recent years (especially since 2015) the number of people living in food insecurity has increased. It will be a huge challenge to achieve the SDGs of zero hunger by 2030 [ 6 ]. FAO et al. [ 7 ] projected that one in four individuals in SSA were undernourished in 2017. Moreover, FAO et al. [ 8 ] found that, between 2014 and 2018, the prevalence of undernourishment worsened. Twenty percent of the continent's population, or 256 million people, are undernourished today, of which 239 million are in SSA. Hidden hunger is also one of the most severe types of malnutrition (micronutrient deficiencies). One in three persons suffers from inadequacies related to hidden hunger, which impacts two billion people worldwide [ 9 ]. Similarly, SSA has a high prevalence of hidden hunger [ 10 , 11 ]. An important consequence of food insecurity is that around 9 million people die yearly worldwide due to hunger and hunger-related diseases. This is more than from Acquired Immunodeficiency Syndrome (AIDS), malaria, and tuberculosis combined [ 6 ]. Even though the hunger crisis affects many people of all genders and ages, children are particularly affected in Africa. There are too many malnourished children in Africa, and malnutrition is a major factor in the high infant mortality rates and causes physical and mental development delays and disorders in SSA [ 12 ]. According to UN statistics, chronic malnutrition globally accounts for 165 million stunted or underweight children. Around 75% of these kids are from SSA and South Asia. Forty percent of children in SSA are impacted. In SSA, about 3.2 million children under the age of five dies yearly, which is about half of all deaths in this age group worldwide. Malnutrition is responsible for almost one child under the age of five dying every two minutes worldwide. The child mortality rate in the SSA is among the highest in the world, about one in nine children pass away before the age of five [ 12 ]. In addition to the direct impact of food insecurity on health outcomes, it also indirectly contributes to disordered eating patterns, higher or lower blood cholesterol levels, lower serum albumin, lower hemoglobin, vitamin A levels, and poor physical and mental health [ 13 , 14 , 15 ]. Iodine, iron, and zinc deficiency are the most often identified micronutrient deficiencies across all age groups. A deficiency in vitamin A affects an estimated 190 million pre-schoolers and 19 million pregnant women [ 16 ]. Even though it is frequently noted that hidden hunger mostly affects pregnant women, children, and teenagers, it further affects people’s health at all stages of life [ 17 ]. With the above information, researchers and policymakers should focus on the issue of food insecurity and health status. The SDGs that were developed in 2015 intend to end hunger in 2030 as one of its primary targets. However, a growing number of people live with hunger and food insecurity, leading to millions of deaths. Hence, this study questioned what is the impact of food insecurity on people's health outcomes in SSA countries. In addition, despite the evidence implicating food insecurity and poor health status, there is a lack of macro-level empirical studies concerning the impact of food insecurity on people’s health status in SSA countries, which leads to a knowledge (literature) gap. Therefore, this study aims to examine the impact of food insecurity on life expectancy and infant mortality in SSA countries for the period ranging from 2001–2018 using panel mean regression approaches. Theoretical and conceptual frameworkStructural factors, such as climate, socio-economic, social, and local food availability, affect people’s food security. People’s health condition is impacted by food insecurity through nutritional, mental health, and behavioral channels [ 18 ]. Under the nutritional channel, food insecurity has an impact on total caloric intake, diet quality, and nutritional status [ 19 , 20 , 21 ]. Hunger and undernutrition may develop when food supplies are scarce, and these conditions may potentially lead to wasting, stunting, and immunological deficiencies [ 22 ]. However, food insecurity also negatively influences health due to its effects on obesity, women's disordered eating patterns [ 23 ], and poor diet quality [ 24 ]. Under the mental health channel, Whitaker et al. [ 25 ] noted that food insecurity is related to poor mental health conditions (stress, sadness, and anxiety), which have also been linked to obesity and cardiovascular risk [ 26 ]. The effects of food insecurity on mental health can worsen the health of people who are already sick as well as lead to disease acquisition [ 18 ]. Similarly, the behavioral channel argues that there is a connection between food insecurity and health practices that impact disease management, prevention, and treatment. For example, lack of access to household food might force people to make bad decisions that may raise their risk of sickness, such as relying too heavily on cheap, calorically dense, nutrient-poor meals or participating in risky sexual conduct. In addition, food insecurity and other competing demands for survival are linked to poorer access and adherence to general medical treatment in low-income individuals once they become sick [ 27 , 28 , 29 , 30 ] Food insecurity increases the likelihood of exposure to HIV and worsens the health of HIV-positive individuals [ 18 ]. Weiser et al. [ 31 ] found that food insecurity increases the likelihood of unsafe sexual activities, aggravating the spread of HIV. It can also raise the possibility of transmission through unsafe newborn feeding practices and worsening maternal health [ 32 ]. In addition, food insecurity has been linked to decreased antiretroviral adherence, declines in physical health status, worse immunologic status [ 33 ], decreased viral suppression [ 34 , 35 ], increased incidence of serious illness [ 36 ], and increased mortality [ 37 ] among people living with HIV. With the above theoretical relationship between target variables and since this study focuses on the impact of food insecurity on health outcomes, and not on the causes, it adopted the conceptual framework of Weiser et al. [ 18 ] and constructed Fig. 1 . ![what is an outcome variable in research figure 1](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12889-023-15244-3/MediaObjects/12889_2023_15244_Fig1_HTML.png) A conceptual framework of food insecurity and health. Source: Modified and constructed by the author using Weiser et al. [ 18 ] conceptual framework. Permission was granted by Taylor & Francis to use their original Figs. (2.2, 2.3, and 2.4); to develop the above figure. Permission number: 1072954 Several findings associate food insecurity with poorer health, worse disease management, and a higher risk of premature mortality even though they used microdata. For instance, Stuff et al. [ 38 ] found that food insecurity is related to poor self-reported health status, obesity [ 39 ], abnormal blood lipids [ 40 ], a rise in diabetes [ 24 , 40 ], increased gestational diabetes[ 41 ], increased perceived stress, depression and anxiety among women [ 25 , 42 ], Human Immunodeficiency Virus (HIV) acquisition risk [ 43 , 44 , 45 ], childhood stunting [ 46 ], poor health [ 47 ], mental health and behavioral problem [ 25 , 48 , 49 ]. The above highlight micro-level empirical studies, and since the scope of this study is macro-level, Table 1 provides only the existing macro-level empirical findings related to the current study. Empirical findings in Table 1 are a few, implying a limited number of macro-level level empirical findings. Even the existing macro-level studies have several limitations. For instance, most studies either employed conventional estimation techniques or overlooked basic econometric tests; thus, their results and policy implications may mislead policy implementers. Except for Hameed et al. [ 53 ], most studies’ data are either outdated or unbalanced; hence, their results and policy implications may not be valuable in the dynamic world and may not be accurate like balanced data. Besides, some studies used limited (one) sampled countries; however, few sampled countries and observations do not get the asymptotic properties of an estimator [ 56 ]. Therefore, this study tries to fill the existing gaps by employing robust estimation techniques with initial diagnostic and post-estimation tests, basic panel econometric tests and robustness checks, updated data, a large number of samples. Study setting and participantsAccording to Smith and Meade [ 57 ], the highest rates of both food insecurity and severe food insecurity were found in Sub-Saharan Africa in 2017 (55 and 28%, respectively), followed by Latin America and the Caribbean (32 and 12%, respectively) and South Asia (30 and 13%). Similarly, SSA countries have worst health outcomes compared to other regions. For instance, in 2020, the region had the lowest life expectancy [ 58 ] and highest infant mortality [ 59 ]. Having the above information, this study's target population are SSA countries chosen purposively. However, even though SSA comprises 49 of Africa's 55 countries that are entirely or partially south of the Sahara Desert. This study is conducted for a sample of 31 SSA countries (Angola, Benin, Botswana, Burkina Faso, Cameroon, Cabo Verde, Chad, Congo Rep., Côte d'Ivoire, Ethiopia, Gabon, The Gambia, Ghana, Kenya, Lesotho, Liberia, Madagascar, Malawi, Mali, Mauritania, Mauritius, Mozambique, Namibia, Nigeria, Rwanda, Senegal, Sierra Leone, South Africa, Sudan, Tanzania, and Togo). The sampled countries are selected based on data accessibility for each variable included in the empirical models from 2001 to 2018. Since SSA countries suffer from food insecurity and related health problems, this study believes the sampled countries are appropriate and represent the region. Moreover, since this study included a large sample size, it improves the estimator’s precision. Data type, sources, and scopeThis study uses secondary data collected in December 2020 online from the databases of the Food and Agricultural Organization (FAO), the United Nations Development Programme (UNDP), and the World Bank (WB) (see Table 2 ). In addition, the study uses yearly balanced data from 2001 to 2018, which is appropriate because it captures the Millennium Development Goals, SDGs, and other economic conditions, such as the rise of SSA countries’ economies and the global financial crisis of the 2000s. Therefore, this study considers various global development programs and events. Generally, the scope of this study (sampled countries and time) is sufficient to represent SSA countries. In other words, the study has n*T = 558 observations, which fulfills the large sample size criteria recommended by Kennedy [ 56 ]. The empirical modelModel specification is vital to conduct basic panel data econometric tests and estimate the relationship of target variables. Besides social factors, the study includes economic factors determining people's health status. Moreover, it uses two proxies indicators to measure both food insecurity and health status; hence, it specifies the general model as follows: The study uses four models to analyze the impact of food insecurity on health outcomes. where LNLEXP and LNINFMOR (dependent variables) refer to the natural logarithm of life expectancy at birth and infant mortality used as proxy variables for health outcomes. Similarly, PRUND and AVRDES are the prevalence of undernourishment and average dietary energy supply adequacy – proxy and predictor variables for food insecurity. Moreover, to regulate countries’ socio-economic conditions and to account for time-varying bias that can contribute to changes in the dependent variable, the study included control variables, such as GDPPC, GOVEXP, MNSCHOOL, and URBAN. GDPPC is GDP per capita, GOVEXP refers to domestic general government health expenditure, MNSCHOOL is mean years of schooling and URBAN refers to urbanization. Further, n it , v it , ε it , and μ it are the stochastic error terms at period t. The parameters \({\alpha }_{0}, { \beta }_{0}, { \theta }_{0},{ \delta }_{0}\) refer to intercept terms and \({\alpha }_{1}-{\alpha }_{5}, {\beta }_{1}-{\beta }_{5}, { \theta }_{1}-{\theta }_{5}, and {\delta }_{1}-{\delta }_{5}\) are the long-run estimation coefficients. Since health outcomes and food insecurity have two indicators used as proxy variables, this study estimates different alternative models and robustness checks of the main results. Furthermore, the above models did not address heterogeneity problems; hence, this study considers unobserved heterogeneity by introducing cross-section and time heterogeneity in the models. This is accomplished by assuming a two-way error component for the disturbances with: From Eq. 2 , the unobservable individual (cross-section) and unobservable time heterogeneities are described by \({\delta }_{i} and {\tau }_{t}\) (within components), respectively. Nonetheless, the remaining random error term is \({\gamma }_{it}\) (panel or between components). Therefore, the error terms in model 1A-1D will be substituted by the right-hand side elements of Eq. 2 . Depending on the presumptions of whether the error elements are fixed or random, the FE and RE models are the two kinds of models that will be evaluated. Equation ( 2 ) yields a two-way FE error component model, or just a FE model if the assumptions are that \({\delta }_{i} and {\tau }_{t}\) are fixed parameters to be estimated and that the random error component, \({\gamma }_{it}\) , is uniformly and independently distributed with zero mean and constant variance (homoscedasticity). Equation ( 2 ), on the other hand, provides a two-way RE error component model or a RE model if we suppose \({\delta }_{i} and {\tau }_{t}\) are random, just like the random error term, or \({\delta }_{i},{\tau }_{t}, and {\gamma }_{it}\) are all uniformly and independently distributed with zero mean and constant variance, or they are all independent of each other and independent variables [ 60 ]. Rather than considering both error components, \({\delta }_{i}, and {\tau }_{t}\) , we can examine only one of them at a time (fixed or random), yielding a one-way error component model, FE or RE. The stochastic error term \({\varpi }_{it}\) in Eq. 2 will then be: Statistical analysisThis study conducted descriptive statistics, correlation analysis, and initial diagnosis tests (cross-sectional and time-specific fixed effect, outliers and influential observations, multicollinearity, normality, heteroscedasticity, and serial correlation test). Moreover, it provides basic panel econometric tests and panel data estimation techniques. For consistency, statistical software (STATA) version 15 was used for all analyses. Descriptive statistics and correlation analysisDescriptive statistics is essential to know the behavior of the variables in the model. Therefore, it captures information, such as the mean, standard deviation, minimum, maximum, skewness, and kurtosis. Similarly, the study conducted Pearson correlation analysis to assess the degree of relationship between the variables. Initial diagnosisCross-sectional and time-specific fixed effect. One can anticipate differences arising over time or within the cross-sectional units, given that the panel data set comprises repeated observations over the same units gathered over many periods. Therefore, before estimation, this study considered unexplained heterogeneity in the models. One fundamental limitation of cross-section, panel, and time series data regression is that they do not account for country and time heterogeneity [ 60 ]. These unobserved differences across nations and over time are crucial in how the error term is represented and the model is evaluated. These unobserved heterogeneities, however, may be represented by including both country and time dummies in the regression. However, if the parameters exceed the number of observations, the estimate will fail [ 60 ]. However, in this study, the models can be estimated. If we include both country and time dummies, we may assume that the slope coefficients are constant, but the intercept varies across countries and time, yielding the two-way error components model. As a result, this study examines the null hypothesis that intercepts differ across nations and time in general. Detecting outliers and influential observationsIn regression analysis, outliers and influential observations may provide biased findings. Therefore, the Cooks D outlier and influential observation test was used in the study to handle outliers and influencing observations. To evaluate whether these outliers have a stronger impact on the model to be estimated, each observation in this test was reviewed and compared with Cook’s D statistic [ 61 ]. Cook distance evaluates the extent to which observation impacts the entire model or the projected values. Hence, this study tested the existence of outliers. Normality, heteroscedasticity, multicollinearity, and serial correlation testBefore the final regression result, the data used for the variables were tested for normality, heteroscedasticity, multicollinearity, and serial correlation to examine the characteristics of the sample. Regression models should be checked for nonnormal error terms because a lack of Gaussianity (normal distribution) can occasionally compromise the accuracy of estimation and testing techniques. Additionally, the validity of inference techniques, specification tests, and forecasting critically depends on the normalcy assumption [ 62 ]. Similarly, multicollinearity in error terms leads to a dataset being highly sensitive to a minor change, instability in the regression model, and skewed and unreliable results. Therefore, this study conducted the normality using Alejo et al. [ 62 ] proposed command and multicollinearity (using VIF) tests. Most conventional panel data estimation methods rely on homoscedastic individual error variance and constant serial correlation. Since the error component is typically connected to the variance that is not constant during the observation and is serially linked across periods, these theoretical presumptions have lately reduced the applicability of various panel data models. Serial correlation and heteroskedasticity are two estimate issues frequently connected to cross-sectional and time series data, respectively. Similarly, panel data is not free from these issues because it includes cross-sections and time series, making the estimated parameters ineffective, and rendering conclusions drawn from the estimation incorrect [ 63 ]. Therefore, this study used the Wooldridge [ 63 ] test for serial correlation in linear panel models as well as the modified Wald test for heteroskedasticity. Basic panel econometric testsThe basic panel data econometric tests are prerequisites for estimating the panel data. The three main basic panel data tests are cross-sectional dependence, unit root, and cointegration. Cross-sectional dependence (CD)A growing body of the panel data literature concludes that panel data models are likely to exhibit substantial CD in the errors resulting from frequent shocks, unobserved components, spatial dependence, and idiosyncratic pairwise dependence. Even though the impact of CD in estimation depends on several factors, relative to the static model, the effect of CD in dynamic panel estimators is more severe [ 64 ]. Moreover, Pesaran [ 65 ] notes that recessions and economic or financial crises potentially affect all countries, even though they might start from just one or two countries. These occurrences inevitably introduce cross-sectional interdependencies across the cross-sectional unit, their regressors, and the error terms. Hence, overlooking the CD in panel data leads to biased estimates and spurious results [ 64 , 66 ]. Further, the CD test determines the type of panel unit root and cointegration tests we should apply. Therefore, examining the CD is vital in panel data econometrics. In the literature, there are several tests for CD, such as the Breusch and Pagan [ 67 ] Lagrange multiplier (LM) test, Pesaran [ 68 ] scaled LM test, Pesaran [ 68 ] CD test, and Baltagi et al. [ 69 ] bias-corrected scaled LM test (for more detail, see Tugcu and Tiwari [ 70 ]). Besides, Friedman [ 71 ] and Frees [ 72 , 73 ] also have other types of CD tests (for more detail, see De Hoyos and Sarafidis [ 64 ]). This study employs Frees [ 72 ] and Pesaran [ 68 ] among the existing CD tests. This is because, unlike the Breusch and Pagan [ 67 ] test, these tests do not require infinite T and fixed N, and are rather applicable for both a large N and T. Additionally, Free’s CD test can overcome the irregular signs associated with correlation. However, it also employs Friedman [ 71 ] CD for mixed results of the above tests. Unit root testThe panel unit root and cointegration tests are common steps following the CD test. Generally, there are two types of panel unit root tests: (1) the first-generation panel unit root tests, such as Im et al. [ 74 ], Maddala and Wu [ 75 ], Choi [ 76 ], Levin et al. [ 77 ], Breitung [ 78 ] and Hadri [ 79 ], and (2) the second-generation panel unit root tests, such as [ 66 , 80 , 81 , 82 , 83 , 84 , 85 , 86 , 87 , 88 , 89 ]. The first-generation panel unit root tests have been criticized because they assume cross-sectional independence [ 90 , 91 , 92 , 93 ]. This hypothesis is somewhat restrictive and unrealistic, as macroeconomic time series exhibit significant cross-sectional correlation among countries in a panel [ 92 ], and co-movements of economies are often observed in the majority of macroeconomic applications of unit root tests [ 91 ]. The cross-sectional correlation of errors in panel data applications in economics is likely to be the rule rather than the exception [ 93 ]. Moreover, applying first-generation unit root tests under CD models can generate substantial size distortions [ 90 ], resulting in the null hypothesis of nonstationary being quickly rejected [ 66 , 94 ]. As a result, second-generation panel unit root tests have been proposed to take CD into account. Therefore, among the existing second-generation tests, this study employs Pesaran’s [ 66 ] cross-sectionally augmented panel unit root test (CIPS) for models 1A–1C . The rationale for this is that, unlike other unit root tests that allow CD, such as Bai and Ng [ 80 ], Moon and Perron [ 87 ], and Phillips and Sul [ 84 ], Pesaran’s [ 66 ] test is simple and clear. Besides, Pesaran [ 66 ] is robust when time-series’ heteroscedasticity is observed in the unobserved common factor [ 95 ]. Even though theoretically, Moon and Perron [ 87 ], Choi [ 96 ] and Pesaran [ 66 ] require large N and T, Pesaran [ 66 ] is uniquely robust in small sample sizes [ 97 ]. Therefore, this study employs the CIPS test to take into account CD, and heteroskedasticity in the unobserved common factor and both large and small sample countries. However, since there is no CD in model 1D , this study employs the first-generation unit root tests called Levin, Lin, and Chu (LLC), Im, Pesaran, Shin (IPS) and Fisher augmented Dickey–Fuller (ADF) for model 1D . Cointegration testThe most common panel cointegration tests when there is CD are Westerlund [ 98 ], Westerlund and Edgerton [ 99 ], Westerlund and Edgerton [ 100 ], Groen and Kleibergen [ 101 ], Westerlund’s [ 102 ] Durbin-Hausman test, Gengenbach et al. [ 103 ] and Banerjee and Carrion-i-Silvestre [ 104 ]. However, except for a few, most tests are not coded in Statistical Software (STATA) and are affected by insufficient observations. The current study primarily uses Westerlund [ 98 ] and Banerjee and Carrion-i-Silvestre [ 104 ] for models 1A–1C . However, to decide uncertain results, it also uses McCoskey and Kao [ 105 ] cointegration tests for model 1C . The rationale for using Westerlund’s [ 98 ] cointegration test is that most panel cointegration has failed to reject the null hypothesis of no cointegration due to the failure of common-factor restriction [ 106 ]. However, Westerlund [ 98 ] does not require any common factor restriction [ 107 ] and allows for a large degree of heterogeneity (e.g., individual-specific short-run dynamics, intercepts, linear trends, and slope parameters) [ 92 , 107 , 108 ]. Besides, its command is coded and readily available in STATA. However, it suffers from insufficient observations, especially when the number of independent variables increases. The present study employs the Banerjee and Carrion-i-Silvestre [ 104 ] and McCoskey and Kao [ 105 ] cointegration tests to overcome this limitation. The two Engle-Granger-based cointegration tests applicable when there is no CD and are widely used and available in STATA are Pedroni [ 109 , 110 ] and Kao [ 111 ]. However, the Pedroni test has two benefits over Kao: it assumes cross-sectional dependency and considers heterogeneity by employing specific parameters [ 112 ]. Hence, this study uses the Pedroni cointegration test for model 1D . Panel data estimation techniquesThe panel data analysis can be conducted using different estimation techniques and is mainly determined by the results of basic panel econometric tests. Thus, this study mainly employs the Driscoll-Kraay [ 113 ] standard error (DKSE) (for models 1A and 1B ), FE (for model 1C ), and two-step GMM (for model 1D ) estimation techniques to examine the impact of food insecurity on health outcomes. It also employs the Granger causality test. However, for robustness checks, it uses fully modified ordinary least squares (FMOLS), panel-corrected standard error (PCSE), and feasible generalized least squares (FGLS) methods (for models 1A and 1B ). Moreover, it uses a random effect (RE) for model 1C and panel dynamic fixed effect (DFE) techniques for model 1D . Even though several panel estimation techniques allow CD, most of them – such as cross-section augmented autoregressive distributed lag (CS-ARDL), cross-section augmented distributed lag (CS-DL), common correlated effects pooled (CCEP), and common correlated effects mean group (CCEMG) estimators – require a large number of observations over groups and periods. Similarly, the continuously updated fully modified (CUP-FM) and continuously updated bias-corrected (CUP-BC) estimators are not coded in STATA. Others, like the PCSE, FGLS, and seemingly unrelated regression (SUR), are feasible for T (the number of time series) > N (the number of cross-sectional units) [ 114 , 115 ]. However, a DKSE estimate is feasible for N > T [ 114 ]. Therefore, depending on the CD, cointegration test, availability in STATA, and comparing N against T, this study mainly employs the DKSE regression for models 1A and 1B , FE model for model 1C , and GMM for model 1C . Finally, to check the robustness of the main result, this study employs FMOLS, FGLS, and PCSE estimation techniques for models 1A and 1B . Furthermore, even though the Hausman test confirms that the FE is more efficient, the study employs the RE for model 1C . This is because Firebaugh et al. [ 116 ] note that the RE and FE models perform best in panel data. Besides, unlike FE, RE assumes that individual differences are random. In addition, this study uses panel DFE for model 1D (selected based on the Hausman test). Finally, the robustness check is also conducted using an alternative model (i.e., when a dependent variable is without a natural log and Granger causality test). Table 3 shows the overall mean of LNLEXP of the region is 4.063 years which indicates that the region can achieve only 57.43 (using ln(x) = 4.063 = loge (x) = e 4.063 , where e = 2.718) years of life expectancy. This is very low compared to other regions. Besides, the ranges in the value of LNLEXP are between 3.698 and 4.345 or (40–76 years), implying high variation. Similarly, the mean value of LNINFMOR is 3.969; implying SSA countries recorded 52 infants death per 1000. Moreover, the range of LNINFMOR is between 2.525 and 4.919 or (12 – 135 infant death per 1000), implying high variation within the region. The mean value of people’s prevalence for undernourishment is 21.26; indicating 21% of the population is undernourished. However, the mean value of AVRDES is 107.826, which is greater than 100, implying that the calorie supply is adequate for all consumers if the food is distributed according to the requirements of individuals. When we observe the skewness and kurtosis of the variables of the models, except for LNLEXP and LNINFMOR, all variables are positively skewed. In addition, all variables have positive kurtosis with values between 2.202 and 6.092. Table 3 also shows the degree of relationship between variables, such that most values are below the threshold or rule of thumb (0.7) for a greater association [ 117 ]. However, the association between LNINFMOR and LNLEXP, as well as between PRUNP and AVRDES, is over the threshold and seems to have a multicollinearity issue. Nevertheless, these variables did not exist together in the models, indicating the absence of a multicollinearity problem. Table 4 shows whether the cross-sectional specific and time-specific FE in extended models ( model 1A-1D plus Eq. 2 ) are valid. The result reveals that the null hypothesis of the captured unobserved heterogeneity is homogenous across the countries, and time is rejected at 1%, implying the extended models are correctly specified. Besides, to check the robustness of the two-way error component model relative to the pooled OLS estimator, this study conducted an additional poolability test. The result shows the null hypothesis that intercepts homogeneity (pooling) is rejected at a 1% level; thus, the FE model is most applicable, but the pooled OLS is biased. Cooks D is an indicator of high leverage and residuals. The impact is high when D exceeds 4/N, (N = number of observations). A D > 1 implies a significant outlier problem. The Cooks D result of this study confirms the absence of outliers' problem (see supplementary file 1 ). Normality, heteroscedasticity, serial correlation, and multicollinearity testsThe results in Table 5 indicate that the probability value of the joint test for normality on e and u are above 0.01, implying that the residuals are normally distributed. The heteroscedasticity results show that the probability value of the chi-square statistic is less than 0.01 in all models. Therefore, the null hypothesis of constant variance can be rejected at a 1% level of significance. In other words, the modified Wald test result for Groupwise heteroskedasticity presented in Table 5 , rejects the null hypothesis of Groupwise homoskedasticity observed by the probability value of 0.0000, which implies the presence of heteroscedasticity in the residuals. Similarly, all models suffer from serial correlation since the probability value of 0.0000 rejects the null hypothesis of no first-order serial correlation, indicating the presence of autocorrelation in all panel models. Finally, the multicollinearity test reveals that the models have no multicollinearity problem since the Variance inflation Factors (VIF) values are below 5. Cross-sectional dependence testResults in Table 6 strongly reject the null hypothesis of cross-sectional independence for models 1A – 1C . However, for model 1D , the study found mixed results (i.e., Pesaran [ 68 ] fails to reject the null hypothesis of no CD while Frees [ 72 ] strongly rejects it). Thus, to decide, this study employs the Friedman [ 71 ] CD test. The result fails to reject the null hypothesis of cross-sectional independence, implying that two out of three tests fail to reject the null hypothesis of cross-sectional independence in model 1D . Therefore, unlike others, there is no CD in model 1D (see Table 6 ). Unit root testsTable 7 shows that all variables are highly (at 1% level) significant either at level (I(0)) or first difference (I(1)), which implies all variables are stationary. In other words, the result fails to reject the null hypothesis of unit root (non-stationary) for all variables at a 1%-significance level, either at levels or the first differences. Thus, we might expect a long-run connection between these variables collectively. Cointegration testsThe results in Table 8 show that both the Westerlund [ 98 ] and Banerjee and Carrion-i-Silvestre [ 104 ] cointegration tests strongly reject the null hypothesis of no-cointegration in models 1A and 1B . However, model 1C provides a mixed result, i.e. the Banerjee and Carrion-i-Silvestre [ 104 ] test rejects the null hypothesis of no cointegration, yet the reverse is true for the Westerlund [ 98 ] test. Therefore, this study conducted further cointegration tests for model 1C . Even though Westerlund and Edgerton [ 99 ] suffer from insufficient observation, it is based on the McCoskey and Kao [ 105 ] LM test [ 118 ]. Thus, we can use a residual-based cointegration test in the heterogeneous panel framework proposed by McCoskey and Kao [ 105 ]. However, an efficient estimation technique of cointegrated variables is required, and hence the FMOLS and DOLS estimators are recommended. The residuals derived from the FMOLS and DOLS will be tested for stationarity with the null hypothesis of no cointegration amongst the regressors. Since the McCoskey and Kao [ 105 ] test involves averaging the individual LM statistics across the cross-sections, for testing the residuals FMOLS and DOLS stationarity, McCoskey, and Kao [ 105 ] test is in the spirit of IPS (Im et al. [ 74 ]) [ 119 ]. Though FMOLS and DOLS are recommended for the residuals cointegration test, DOLS is better than FMOLS (for more detail, see Kao and Chiang [ 120 ]); therefore, this study uses a residual test derived from DOLS. The result fails to reject the null hypothesis of no cointegration. Two (Banerjee and Carrion-i-Silvestre [ 104 ] and McCoskey and Kao [ 105 ]) out of three tests fail to reject the null hypothesis of no cointegration; hence, we can conclude that there is no long-run relationship among the variables in model 1C . Unlike other models, since there is CD in model 1D , this study employs the Pedroni [ 109 ] and Kao [ 111 ] cointegration tests for model 1D . The result strongly rejects the null hypothesis of no cointegration, which is similar to models 1A and 1B , that a long-run relationship exists among the variables in model 1D (see Table 5 ). Panel data estimation resultsTable 9 provides long-run regression results of all models employing appropriate estimation techniques such as DKSE, FE, and two-step GMM, along with the Granger causality test. However, the DKSE regression can be estimated in three ways: FE with DKSE, RE with DKSE, and pooled Ordinary Least Squares/Weighted Least Squares (pooled OLS/WLS) regression with DKSE. Hence, we must choose the most efficient model using Hausman and Breusch-Pagan LM for RE tests (see supplementary file 2 ). As a result, this study employed FE with DKSE for models 1A and 1B . Further, due to Hausman's result, absence of cointegration and to deal with heterogeneity and spatial dependence in the dynamic panel, this study employs FE for the model1C (see the supplementary file 2). However, due to the absence of CD, the presence of cointegration, and N > T, this study uses GMM for model 1D . Moreover, according to Roodman [ 121 ], the GMM approach can solve heteroskedasticity and autocorrelation problems. Furthermore, even though two-step GMM produces only short-run results, it is possible to generate long-run coefficients from short-run results [ 122 , 123 ]. The DKSE result of model 1A shows that a 1% increment in people's prevalence for undernourishment reduces their life expectancy by 0.00348 PPs (1 year or 366 days). However, in model 1C, a 1% rise in the prevalence of undernourishment increases infant mortality by 0.0119 PPs (1 year or 369 days). The DKSE estimations in model 1B reveal that people’s life expectancy rises by 0.00317 PPs with every 1% increase in average dietary energy supply. However, the GMM result for model 1D confirms that a 1% incrementin average dietary energy supply reduces infant mortality by 0.0139 PPs. Moreover, this study conducted a panel Granger causality test to confirm whether or not food insecurity has a potential causality to health outcomes. The result demonstrates that the null hypothesis of change in people’s prevalence for undernourishment and average dietary energy supply does not homogeneously cause health outcomes is rejected at 1% significance, implying a change in food insecurity does Granger-cause health outcomes of SSA countries (see Table 9 ). In addition to the main results, Table 9 also reports some post-estimation statistics to ascertain the consistency of the estimated results. Hence, in the case of DKSE and FE models, the validity of the models is determined by the values of R 2 and the F statistics. For instance, R 2 quantifies the proportion of the variance in the dependent variable explained by the independent variables, representing the model’s quality. The results in Table 9 demonstrate that the explanatory variables explain more than 62% of the variance on the dependent variable. Cohen [ 125 ] classifies the R 2 value of 2% as a moderate influence in social and behavioral sciences, while 13 and 26% are considered medium and large effects, respectively. Therefore, the explanatory variables substantially impact this study's models. Similarly, the F statistics explain all independent variables jointly explain the dependent one. For the two-step system GMM, the result fails to reject the null hypothesis of no first (AR(1)) and second-order (AR(2)) serial correlation, indicating that there is no first and second-order serial correlation. In addition, the Hansen [ 126 ] and Sargan [ 127 ] tests fail to reject the null hypothesis of the overall validity of the instruments used, which implies too many instruments do not weaken the model. Robustness checksThe author believes the above findings may not be enough for policy recommendations unless robustness checks are undertaken. Hence, the study estimated all models without the natural logarithm of the dependent variables (see Table 10 ). The model 1A result reveals, similar to the above results, individuals’ prevalence for undernourishment significantly reduces their life expectancy in SSA countries. That means a 1% increase in the people's prevalence of undernourishment reduces their life expectancy by 0.1924 PPs. Moreover, in model 1B , life expectancy rises by 0.1763 PPs with every 1% increase in average dietary energy supply. In model 1C , the rise in infants’ prevalence for undernourishment has a positive and significant effect on their mortality rate in SSA countries. The FE result implies that a 1% rise in infants’ prevalence for undernourishment increases their mortality rate by 0.9785 PPs. The GMM result in model 1D indicates that improvement in average dietary energy supply significantly reduces infant mortality. Further, the Granger causality result confirms that the null hypothesis of change in the prevalence of undernourishment and average dietary energy supply does not homogeneously cause health outcomes and is rejected at a 1% level of significance. This implies a change in food insecurity does Granger-cause health outcomes in SSA countries (see Table 10 ). The study also conducted further robustness checks using the same dependent variables (as Table 9 ) but different estimation techniques. The results confirm that people’s prevalence of undernourishment has a negative and significant effect on their life expectancy, but improvement in average dietary energy supply significantly increases life expectancy in SSA countries. However, the incidence of undernourishment in infants contributes to their mortality; however, progress in average dietary energy supply for infants significantly reduces their mortality (see Table 11 ). The main objective of this study is to examine the impact of food insecurity on the health outcomes of SSA countries. Accordingly, the DKSE result of model 1A confirms that the rise in people’s prevalence for undernourishment significantly reduces their life expectancy in SSA countries. However, the FE result shows that an increment in the prevalence of undernourishment has a positive and significant impact on infant mortality in model 1C . This indicates that the percentage of the population whose food intake is insufficient to meet dietary energy requirements is high, which leads to reduce life expectancy but increases infant mortality in SSA countries. The reason for this result is linked to the insufficient food supply in SSA due to low production and yields, primitive tools, lack of supporting smallholder farms and investment in infrastructure, and government policies. Besides, even though the food is available, it is not distributed fairly according to the requirements of individuals. Moreover, inadequate access to food, poor nutrition, and chronic illnesses are caused by a lack of well-balanced diets. In addition, many of these countries are impacted by poverty, making it difficult for citizens to afford nutritious food. All these issues combine to create an environment where individuals are more likely to suffer malnutrition-related illnesses, resulting in a lower life expectancy rate. The DKSE estimation result in model 1B reveals that improvement in average dietary energy supply positively impacts people's life expectancy in SSA countries. However, the improvement in average dietary energy supply reduces infant mortality. Based on the above results, we can conclude that food insecurity harms SSA nations' health outcomes. This is because the prevalence of undernourishment leads to increased infant mortality by reducing the vulnerability, severity, and duration of infectious diseases such as diarrhea, pneumonia, malaria, and measles. Similarly, the prevalence of undernourishment can reduce life expectancy by increasing the vulnerability, severity, and duration of infectious diseases. However, food security improves health outcomes – the rise in average dietary energy supply reduces infant mortality and increases the life expectancy of individuals. Several facts and theories support the above findings. For instance, similar to the theoretical and conceptual framework section, food insecurity in SSA countries can affect health outcomes in nutritional, mental health, and behavioral channels. According to FAO et al. [ 128 ], the prevalence of undernourishment increased in Africa from 17.6% of the population in 2014 to 19.1% in 2019. This figure is more than twice the global average and the highest of all regions of the world. Similarly, SSA is the world region most at risk of food insecurity [ 129 ]. According to Global Nutrition [ 130 ] report, anemia affects an estimated 39.325% of women of reproductive age. Some 13.825% of infants have a low weight at birth in the SSA region. Excluding middle African countries (due to lack of data), the estimated average prevalence of infants aged 0 to 5 months who are exclusively breastfed is 35.73%, which is lower than the global average of 44.0%. Moreover, SSA Africa still experiences a malnutrition burden among children aged under five years. The average prevalence of overweight is 8.15%, which is higher than the global average of 5.7%. The prevalence of stunting is 30.825%—higher than the worldwide average of 22%. Conversely, the SSA countries’ prevalence of wasting is 5.375%, which is higher than most regions such as Central Asia, Eastern Asia, Western Asia, Latin America and the Caribbean, and North America. The SSA region's adult population also faces a malnutrition burden: an average of 9.375% of adult (aged 18 and over) women live with diabetes, compared to 8.25% of men. Meanwhile, 20.675% of women and 7.85% of men live with obesity. According to Saltzman et al. [ 17 ], micronutrient deficiencies can affect people’s health throughout their life cycle. For instance, at the baby age, it causes (low birth weight, higher mortality rate, and impaired mental development), child (stunting, reduced mental capacity, frequent infections, reduced learning capacity, higher mortality rate), adolescent (stunting, reduced mental capacity, fatigue, and increased vulnerability to infection), pregnant women (increased mortality and perinatal complications), adult (reduced productivity, poor socio-economic status, malnutrition, and increased risk of chronic disease), elderly (increased morbidity (including osteoporosis and mental impairment), and higher mortality rate). Though this study attempts to fill the existing gaps, it also has limitations. It examined the impact of food insecurity on infant mortality; however, their association is reflected indirectly through other health outcomes. Hence, future studies can extend this study by examining the indirect effect of food insecurity on infant mortality, which helps to look at in-depth relationships between the variables. Moreover, this study employed infant mortality whose age is below one year; hence, future studies can broaden the scope by decomposing infant mortality into (neonatal and postnatal) and under-five mortality. Millions of people are dying every year due to hunger and hunger-related diseases worldwide, especially in SSA countries. Currently, the link between food insecurity and health status is on researchers' and policymakers' agendas. However, macro-level findings in this area for most concerned countries like SSA have been given only limited attention. Therefore, this study examined the impact of food insecurity on life expectancy and infant mortality rates. The study mainly employs DKSE, FE, two-step GMM, and Granger causality approaches, along with other estimation techniques for robustness checks for the years between 2001 and 2018. The result confirms that food insecurity harms health outcomes, while food security improves the health status of SSA nations'. That means that a rise in undernourishment increases the infant mortality rate and reduces life expectancy. However, an improvement in the average dietary energy supply reduces infant mortality and increases life expectancy. Therefore, SSA countries need to guarantee their food accessibility both in quality and quantity, which improves health status. Both development experts and political leaders agree that Africa has the potential for agricultural outputs, can feed the continent, and improve socio-economic growth. Besides, more than half of the world's unused arable land is found in Africa. Therefore, effective utilization of natural resources is essential to achieve food security. Moreover, since the majority of the food in SSA is produced by smallholder farmers [ 131 ] while they are the most vulnerable to food insecurity and poverty [ 132 , 133 ]; hence, special focus and support should be given to smallholder farmers that enhance food self-sufficiency. Further, improvement in investment in agricultural research; improvement in markets, infrastructures, and institutions; good macroeconomic policies and political stability; and developing sub-regional strategies based on their agroecological zone are crucial to overcoming food insecurity and improving health status. Finally, filling a stomach is not sufficient; hence, a person's diet needs to be comprehensive and secure, balanced (including all necessary nutrients), and available and accessible. Therefore, SSA countries should ensure availability, accessibility, usability, and sustainability to achieve food and nutrition security. Availability of data and materialsThe datasets used and/or analyzed during the current study are available in supplementary materials. AbbreviationsAugmented Dickey–Fuller Acquired Immunodeficiency Syndrome Average Dietary Energy Supply Common Correlated Effects Mean Group Common Correlated Effects Pooled Cross-Sectional Dependence Cross-Sectionally Augmented Panel Unit Root Test Cross-Section Augmented Autoregressive Distributed Lag Cross-Section Augmented Distributed Lag Continuously Updated Bias-Corrected Continuously Updated Full Modified Dynamic Fixed Effect Driscoll-Kraay Standard Errors Dynamic Ordinary Least Square Error Correction Model Food and Agricultural Organization Fixed Effect Feasible Generalised Least Squares Fully Modified Ordinary Least Square Gross Domestic Product (GDP) per capita Generalised Method of Momentum Domestic General Government Health Expenditure Human Immunodeficiency Virus Integration at First Difference International Fund for Agricultural Development Infant Mortality Rate Im, Pesaran, Shin Lag of Infant Mortality Rate Lag of Natural Logarithm of Infant Mortality Rate Life Expectancy at Birth Levin, Lin, and Chu Lagrange Multiplier Natural Logarithm of Infant Mortality Rate Natural Logarithm of Life Expectancy at Birth Mean Years of Schooling Ordinary Least Squares Panel-Corrected Standard Error Pooled Mean Group Prevalence of Undernourishment Random Effect Sustainable Development Goals Sub-Saharan African Statistical Software Seemingly Unrelated Regression Urbanisation World Food Programme World Health Organization Weighted Least Squares Giller KE. 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Sargan JD. The estimation of economic relationships using instrumental variables. Econometrica. 1958;26(3):393–415. FAO, IFAD, UNICEF, WFP, WHO The State of Food Security and Nutrition in the World. Transforming Food Systems to Deliver Affordable Healthy Diets for All FAO. 2020. Available from: https://www.fao.org/publications/sofi/2020/en/ . Van Ittersum MK, Van Bussel LG, Wolf J, Grassini P, Van Wart J, Guilpart N, Claessens L, De Groot H, Wiebe K, Mason-D’Croz D, Yang H. Can sub-Saharan Africa feed itself? Proce Natl Acad Sci. 2016;113(52):14964–9. Global Nutrition Report. Country Nutrition Profiles. 2021. https://globalnutritionreport.org/resources/nutrition-profiles/africa/ . Herrero M, Thornton PK, Power B, Bogard JR, Remans R, Fritz S, Gerber JS, Nelson G, See L, Waha K, Watson RA. Farming and the geography of nutrient production for human use: a transdisciplinary analysis. Lancet Planet Health. 2017;1(1):e33-42. Sibhatu KT, Qaim M. Rural food security, subsistence agriculture, and seasonality. PLoS ONE. 2017;12(10): e0186406. Fanzo J. The role of farming and rural development is central to our diets. Physiol Behav. 2018;193:291–7. Download references AcknowledgementsThe author sincerely thanks the Editor, the three anonymous reviewers, and BalázsKotosz (Adjunct Professor) for their comments and advice. This study did not receive any specific grant. Author informationAuthors and affiliations. College of Business and Economics, Department of Economics, Arsi University, Asella, Ethiopia Sisay Demissew Beyene You can also search for this author in PubMed Google Scholar ContributionsSDB collected, analyzed, and interpreted the data, and wrote and approved the paper for submission. Corresponding authorCorrespondence to Sisay Demissew Beyene . Ethics declarationsEthics approval and consent to participate. Not applicable. Consent for publicationCompeting interests. The author declares that there are no competing interests. Additional informationPublisher’s note. Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations. Supplementary InformationAdditional file 1:. Table S1. Cook’s D results Additional file 2: Table S2. Hausman and Breusch-Pagan LM for REtests. Additional file 3.Rights and permissions. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. 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What is generative AI? Artificial intelligence that createsGenerative ai models can carry on conversations, answer questions, write stories, produce source code, and create images and videos of almost any description. here's how generative ai works, how it's being used, and why it’s more limited than you might think.. ![what is an outcome variable in research Josh Fruhlinger](https://images.idgesg.net/images/article/2019/05/joshfruhlinger_crop-100796869-byline.jpg?auto=webp&quality=85,70) Contributing writer, InfoWorld | ![what is an outcome variable in research shutterstock 1117048970 colorful balloons flying among paper planes and against a cloudy blue sky](https://images.idgesg.net/images/article/2023/08/shutterstock_1117048970-100944335-large.jpg?auto=webp&quality=85,70) The emergence of generative AIHow does generative ai work, what is an ai model, is generative ai sentient, testing the limits of computer intelligence. - Why does AI art have too many fingers?
- Potential negative impacts of generative AI
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Generative AI is a kind of artificial intelligence that creates new content, including text, images, audio, and video, based on patterns it has learned from existing content. Today’s generative AI models have been trained on enormous volumes of data using deep learning , or deep neural networks, and they can carry on conversations, answer questions, write stories, produce source code, and create images and videos of any description, all based on brief text inputs or “prompts.” Generative AI is called generative because the AI creates something that didn’t previously exist. That’s what makes it different from discriminative AI , which draws distinctions between different kinds of input. To say it differently, discriminative AI tries to answer a question like “Is this image a drawing of a rabbit or a lion?” whereas generative AI responds to prompts like “Draw me a picture of a lion and a rabbit sitting next to each other.” This article introduces you to generative AI and its uses with popular models like ChatGPT and DALL-E . We’ll also consider the limitations of the technology, including why “too many fingers” has become a dead giveaway for artificially generated art. Generative AI has been around for years, arguably since ELIZA , a chatbot that simulates talking to a therapist, was developed at MIT in 1966. But years of work on AI and machine learning have recently come to fruition with the release of new generative AI systems. You’ve almost certainly heard about ChatGPT , a text-based AI chatbot that produces remarkably human-like prose. DALL-E and Stable Diffusion have also drawn attention for their ability to create vibrant and realistic images based on text prompts. Output from these systems is so uncanny that it has many people asking philosophical questions about the nature of consciousness—and worrying about the economic impact of generative AI on human jobs. But while all of these artificial intelligence creations are undeniably big news, there is arguably less going on beneath the surface than some may assume. We’ll get to some of those big-picture questions in a moment. First, let’s look at what’s going on under the hood. Generative AI uses machine learning to process a huge amount of visual or textual data, much of which is scraped from the internet, and then determines what things are most likely to appear near other things. Much of the programming work of generative AI goes into creating algorithms that can distinguish the “things” of interest to the AI’s creators—words and sentences in the case of chatbots like ChatGPT , or visual elements for DALL-E . But fundamentally, generative AI creates its output by assessing an enormous corpus of data, then responding to prompts with something that falls within the realm of probability as determined by that corpus. Autocomplete—when your cell phone or Gmail suggests what the remainder of the word or sentence you’re typing might be—is a low-level form of generative AI. ChatGPT and DALL-E just take the idea to significantly more advanced heights. ChatGPT and DALL-E are interfaces to underlying AI functionality that is known in AI terms as a model. An AI model is a mathematical representation—implemented as an algorithm, or practice—that generates new data that will (hopefully) resemble a set of data you already have on hand. You’ll sometimes see ChatGPT and DALL-E themselves referred to as models; strictly speaking this is incorrect, as ChatGPT is a chatbot that gives users access to several different versions of the underlying GPT model. But in practice, these interfaces are how most people will interact with the models, so don’t be surprised to see the terms used interchangeably. AI developers assemble a corpus of data of the type that they want their models to generate. This corpus is known as the model’s training set, and the process of developing the model is called training . The GPT models, for instance, were trained on a huge corpus of text scraped from the internet, and the result is that you can feed it natural language queries and it will respond in idiomatic English (or any number of other languages, depending on the input). AI models treat different characteristics of the data in their training sets as vectors —mathematical structures made up of multiple numbers. Much of the secret sauce underlying these models is their ability to translate real-world information into vectors in a meaningful way, and to determine which vectors are similar to one another in a way that will allow the model to generate output that is similar to, but not identical to, its training set. There are a number of different types of AI models out there, but keep in mind that the various categories are not necessarily mutually exclusive. Some models can fit into more than one category. Probably the AI model type receiving the most public attention today is the large language models , or LLMs. LLMs are based on the concept of a transformer, first introduced in “ Attention Is All You Need ,” a 2017 paper from Google researchers. A transformer derives meaning from long sequences of text to understand how different words or semantic components might be related to one another, then determines how likely they are to occur in proximity to one another. The GPT models are LLMs, and the T stands for transformer. These transformers are run unsupervised on a vast corpus of natural language text in a process called pretraining (that’s the P in GPT), before being fine-tuned by human beings interacting with the model. Diffusion is commonly used in generative AI models that produce images or video. In the diffusion process, the model adds noise —randomness, basically—to an image, then slowly removes it iteratively, all the while checking against its training set to attempt to match semantically similar images. Diffusion is at the core of AI models that perform text-to-image magic like Stable Diffusion and DALL-E. A generative adversarial network , or GAN, is based on a type of reinforcement learning , in which two algorithms compete against one another. One generates text or images based on probabilities derived from a big data set. The other—a discriminative AI—assesses whether that output is real or AI-generated. The generative AI repeatedly tries to “trick” the discriminative AI, automatically adapting to favor outcomes that are successful. Once the generative AI consistently “wins” this competition, the discriminative AI gets fine-tuned by humans and the process begins anew. One of the most important things to keep in mind here is that, while there is human intervention in the training process, most of the learning and adapting happens automatically. Many, many iterations are required to get the models to the point where they produce interesting results, so automation is essential. The process is quite computationally intensive, and much of the recent explosion in AI capabilities has been driven by advances in GPU computing power and techniques for implementing parallel processing on these chips . The mathematics and coding that go into creating and training generative AI models are quite complex, and well beyond the scope of this article. But if you interact with the models that are the end result of this process, the experience can be decidedly uncanny. You can get DALL-E to produce things that look like real works of art. You can have conversations with ChatGPT that feel like a conversation with another human. Have researchers truly created a thinking machine? Chris Phipps, a former IBM natural language processing lead who worked on Watson AI products, says no. He describes ChatGPT as a “very good prediction machine.” It’s very good at predicting what humans will find coherent. It’s not always coherent (it mostly is) but that’s not because ChatGPT “understands.” It’s the opposite: humans who consume the output are really good at making any implicit assumption we need in order to make the output make sense. Phipps, who’s also a comedy performer, draws a comparison to a common improv game called Mind Meld. Two people each think of a word, then say it aloud simultaneously—you might say “boot” and I say “tree.” We came up with those words completely independently and at first, they had nothing to do with each other. The next two participants take those two words and try to come up with something they have in common and say that aloud at the same time. The game continues until two participants say the same word. Maybe two people both say “lumberjack.” It seems like magic, but really it’s that we use our human brains to reason about the input (“boot” and “tree”) and find a connection. We do the work of understanding, not the machine. There’s a lot more of that going on with ChatGPT and DALL-E than people are admitting. ChatGPT can write a story, but we humans do a lot of work to make it make sense. Certain prompts that we can give to these AI models will make Phipps’ point fairly evident. For instance, consider the riddle “What weighs more, a pound of lead or a pound of feathers?” The answer, of course, is that they weigh the same (one pound), even though our instinct or common sense might tell us that the feathers are lighter. ChatGPT will answer this riddle correctly, and you might assume it does so because it is a coldly logical computer that doesn’t have any “common sense” to trip it up. But that’s not what’s going on under the hood. ChatGPT isn’t logically reasoning out the answer; it’s just generating output based on its predictions of what should follow a question about a pound of feathers and a pound of lead. Since its training set includes a bunch of text explaining the riddle, it assembles a version of that correct answer. However, if you ask ChatGPT whether two pounds of feathers are heavier than a pound of lead, it will confidently tell you they weigh the same amount, because that’s still the most likely output to a prompt about feathers and lead, based on its training set. It can be fun to tell the AI that it’s wrong and watch it flounder in response; I got it to apologize to me for its mistake and then suggest that two pounds of feathers weigh four times as much as a pound of lead. - Artificial Intelligence
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Typical examples of outcomes are cure, clinical worsening, and mortality. The primary outcome is the variable that is the most relevant to answer the research question. Ideally, it should be patient-centered (i.e., an outcome that matters to patients, such as quality of life and survival). Secondary outcomes are additional outcomes monitored to ...
The independent variable is the cause. Its value is independent of other variables in your study. The dependent variable is the effect. Its value depends on changes in the independent variable. Example: Independent and dependent variables. You design a study to test whether changes in room temperature have an effect on math test scores.
The outcome is the attribute that you think might be predicted, or affected, by other attributes - for example, a disease that is affected by lifestyle factors. In a mathematical model, it is normally placed on the left hand side of the equation. The variables that you think might have an effect on the outcome are placed on the right hand ...
Quality of diet is the predictor variable, and health is the outcome variable. 2. Noise Pollution and IQ. One scientist speculates that living in a noisy environment will affect a person's ability to concentrate, which will then affect their mental acuity and subsequent cognitive development.
It is also known as the outcome variable, as it is the variable that is affected by the independent variable. Examples of dependent variables include blood pressure, test scores, and reaction time. ... Scientific research: Variables are used in scientific research to understand the relationships between different factors and to make predictions ...
The Role of Variables in Research. In scientific research, variables serve several key functions: Define Relationships: Variables allow researchers to investigate the relationships between different factors and characteristics, providing insights into the underlying mechanisms that drive phenomena and outcomes. Establish Comparisons: By manipulating and comparing variables, scientists can ...
In an experiment, you manipulate the independent variable and measure the outcome in the dependent variable. For example, in an experiment about the effect of nutrients on crop growth: The independent variable is the amount of nutrients added to the crop field. The dependent variable is the biomass of the crops at harvest time.
Within research, especially scientific research, variables form the foundation of studies, as researchers are often interested in how one variable impacts another, and the relationships between different variables. For example: How someone's age impacts their sleep quality; How different teaching methods impact learning outcomes
All variables can be defined by their Scale of Measurement. Variables in research can also be described by whether the experimenter thinks that they are the cause of a behavior (IV), or the effect (DV). The IV is the variable that you use to do the explaining and the DV is the variable being explained.
The terminology used to distinguish between different roles that a variable can play when analysing a data set. We could also use predictors and outcomes. The idea here is that what you're trying to do is use X X (the predictors) to make guesses about Y Y (the outcomes). This is summarized in the table:
A dependent variable, also called an outcome variable, is the result of the action of one or more independent variables. It can also be defined as any outcome variable associated with some measure, such as a survey. ... Dependent variable. In Encyclopedia of Research Design (Vol. 0, pp. 348-348). SAGE Publications, Inc., https:// doi. org/10. ...
Variable. In most research, one or more outcome variables are measured. Statistical analysis is done on the outcome measures, and conclusions are drawn from the statistical analysis. One common source of misleading research results is giving inadequate attention to the choice of outcome variables. Making a good choice depends on the particulars ...
In experimental research, the independent variable is what differentiates the control group from the experimental group, thereby setting the stage for meaningful comparison and analysis. Dependent variables. Dependent variables are the outcomes or effects that researchers aim to explore and understand in their studies.
1.1.2 - Explanatory & Response Variables. In some research studies one variable is used to predict or explain differences in another variable. In those cases, the explanatory variable is used to predict or explain differences in the response variable. In an experimental study, the explanatory variable is the variable that is manipulated by the ...
Experimental and Non-Experimental Research. Experimental research: In experimental research, the aim is to manipulate an independent variable(s) and then examine the effect that this change has on a dependent variable(s).Since it is possible to manipulate the independent variable(s), experimental research has the advantage of enabling a researcher to identify a cause and effect between variables.
A variable in research simply refers to a person, place, thing, or phenomenon that you are trying to measure in some way. The best way to understand the difference between a dependent and independent variable is that the meaning of each is implied by what the words tell us about the variable you are using. You can do this with a simple exercise ...
A control variable in research is a factor that's kept constant to ensure that it doesn't influence the outcome. By controlling these variables, researchers can isolate the effects of the independent variable on the dependent variable, ensuring that other factors don't skew the results or introduce bias into the experiment.
Outcome research is often characterised by the use of outcome measures, designed to identify the changes that take place during therapy. These contrast with process measures, which aim to identify the variables that cause these changes. One example of an outcome measure is the PHQ-9, which is a self-report measure of depressive symptoms. This can be completed at various intervals throughout ...
Outcome variables are usually the dependent variables which are observed and measured by changing independent variables. These variables determine the effect of the cause (independent) variables when changed for different values. The dependent variables are the outcomes of the experiments determining what was caused or what changed as a result ...
Identify variables. The researcher manipulates the independent variable and the dependent variable is the measured outcome. Operationalized the variables being investigated. Operationalization of a hypothesis refers to the process of making the variables physically measurable or testable, e.g. if you are about to study aggression, you might ...
Food insecurity adversely affects human health, which means food security and nutrition are crucial to improving people's health outcomes. Both food insecurity and health outcomes are the policy and agenda of the 2030 Sustainable Development Goals (SDGs). However, there is a lack of macro-level empirical studies (Macro-level study means studies at the broadest level using variables that ...
Generative AI is a kind of artificial intelligence that creates new content, including text, images, audio, and video, based on patterns it has learned from existing content. Today's generative ...
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